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CITIZENS THREATEN TO ARREST LOCAL JUDGE , LAW ENFORCEMENT ON EDGE

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18 USC 241: Conspiracy against rights Text contains those laws in effect on February 19, 2014

From Title 18-CRIMES AND CRIMINAL PROCEDUREPART I-CRIMESCHAPTER 13-CIVIL RIGHTS

§241. Conspiracy against rights

If two or more persons conspire to injure, oppress, threaten, or intimidate any person in any State, Territory, Commonwealth, Possession, or District in the free exercise or enjoyment of any right or privilege secured to him by the Constitution or laws of the United States, or because of his having so exercised the same; or

If two or more persons go in disguise on the highway, or on the premises of another, with intent to prevent or hinder his free exercise or enjoyment of any right or privilege so secured-

They shall be fined under this title or imprisoned not more than ten years, or both; and if death results from the acts committed in violation of this section or if such acts include kidnapping or an attempt to kidnap, aggravated sexual abuse or an attempt to commit aggravated sexual abuse, or an attempt to kill, they shall be fined under this title or imprisoned for any term of years or for life, or both, or may be sentenced to death.

(June 25, 1948, ch. 645, 62 Stat. 696; Pub. L. 90–284, title I, §103(a), Apr. 11, 1968, 82 Stat. 75; Pub. L. 100–690, title VII, §7018(a), (b)(1), Nov. 18, 1988, 102 Stat. 4396; Pub. L. 103–322, title VI, §60006(a), title XXXII, §§320103(a), 320201(a), title XXXIII, §330016(1)(L), Sept. 13, 1994, 108 Stat. 1970, 2109, 2113, 2147; Pub. L. 104–294, title VI, §§604(b)(14)(A), 607(a), Oct. 11, 1996, 110 Stat. 3507, 3511.)

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Historical and Revision Notes

Based on title 18, U.S.C., 1940 ed., §51 (Mar. 4, 1909, ch. 321, §19, 35 Stat. 1092).

Clause making conspirator ineligible to hold office was omitted as incongruous because it attaches ineligibility to hold office to a person who may be a private citizen and who was convicted of conspiracy to violate a specific statute. There seems to be no reason for imposing such a penalty in the case of one individual crime, in view of the fact that other crimes do not carry such a severe consequence. The experience of the Department of Justice is that this unusual penalty has been an obstacle to successful prosecutions for violations of the act.

Mandatory punishment provision was rephrased in the alternative.

Minor changes in phraseology were made.

Amendments

1996-Pub. L. 104–294, §607(a), substituted “any State, Territory, Commonwealth, Possession, or District” for “any State, Territory, or District” in first par.

Pub. L. 104–294, §604(b)(14)(A), repealed Pub. L. 103–322, §320103(a)(1). See 1994 Amendment note below.

1994-Pub. L. 103–322, §330016(1)(L), substituted “They shall be fined under this title” for “They shall be fined not more than $10,000” in third par.

Pub. L. 103–322, §320201(a), substituted “person in any State” for “inhabitant of any State” in first par.

Pub. L. 103–322, §320103(a)(2)–(4), in third par., substituted “results from the acts committed in violation of this section or if such acts include kidnapping or an attempt to kidnap, aggravated sexual abuse or an attempt to commit aggravated sexual abuse, or an attempt to kill, they shall be fined under this title or imprisoned for any term of years or for life, or both” for “results, they shall be subject to imprisonment for any term of years or for life”.

Pub. L. 103–322, §320103(a)(1), which provided for amendment identical to Pub. L. 103–322, §330016(1)(L), above, was repealed by Pub. L. 104–294, §604(b)(14)(A).

Pub. L. 103–322, §60006(a), substituted “, or may be sentenced to death.” for period at end of third par.

1988-Pub. L. 100–690 struck out “of citizens” after “rights” in section catchline and substituted “inhabitant of any State, Territory, or District” for “citizen” in text.

1968-Pub. L. 90–284 increased limitation on fines from $5,000 to $10,000 and provided for imprisonment for any term of years or for life when death results.

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Effective Date of 1996 Amendment

Amendment by section 604(b)(14)(A) of Pub. L. 104–294 effective Sept. 13, 1994, see section 604(d) of Pub. L. 104–294, set out as a note under section 13 of this title.

Short Title of 1996 Amendment

Pub. L. 104–155, §1, July 3, 1996, 110 Stat. 1392, provided that: “This Act [amending section 247 of this title and section 10602 of Title 42, The Public Health and Welfare, enacting provisions set out as a note under section 247 of this title, and amending provisions set out as a note under section 534 of Title 28, Judiciary and Judicial Procedure] may be cited as the ‘Church Arson Prevention Act of 1996’.”

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Myxobolus cerebralis

From Wikipedia, the free encyclopedia
Myxobolus cerebralis
Triactinomyxon stage of Myxobolus cerebralis – note the three “tails”
Scientific classification e
Kingdom: Animalia
Phylum: Cnidaria
Class: Myxosporea
Order: Bivalvulida
Family: Myxobolidae
Genus: Myxobolus
Species: M. cerebralis
Binomial name
Myxobolus cerebralis
Hofer, 1903
Synonyms
Myxosoma cerebralis
Triactinomyxon dubium
Triactinomyxon gyrosalmo

Myxobolus cerebralis is a myxosporean parasite of salmonids (salmon, trout, and their allies) that causes whirling disease in farmed salmon and trout and also in wild fish populations. It was first described in rainbow trout in Germany a century ago, but its range has spread and it has appeared in most of Europe (including Russia), the United States, South Africa[1] and other countries. In the 1980s, M. cerebralis was found to require a tubificid oligochaete (a kind of segmented worm) to complete its life cycle.[2] The parasite infects its hosts with its cells after piercing them with polar filaments ejected from nematocyst-like capsules.

Whirling disease afflicts juvenile fish (fingerlings and fry) and causes skeletal deformation and neurological damage. Fish “whirl” forward in an awkward, corkscrew-like pattern instead of swimming normally, find feeding difficult, and are more vulnerable to predators. The mortality rate is high for fingerlings, up to 90% of infected populations, and those that do survive are deformed by the parasites residing in their cartilage and bone. They act as a reservoir for the parasite, which is released into water following the fish’s death. M. cerebralis is one of the most economically important myxozoans in fish, as well as one of the most pathogenic. It was the first myxosporean whose pathology and symptoms were described scientifically.[3] The parasite is not transmissible to humans.

Taxonomy

The taxonomy and naming of both M. cerebralis, and of myxozoans in general, have complicated histories. It was originally thought to infect fish brains (hence the specific epithet cerebralis) and nervous systems, it soon was found to primarily infect cartilage and skeletal tissue. Attempts to change the name to Myxobolus chondrophagus, which would more accurately describe the organism, failed because nomenclature rules.[1] Later, the organisms previously called Triactinomyxon dubium and T. gyrosalmo (class Actinosporea) were found to be, in fact, triactinomyxon stages of M. cerebralis, the life cycle of which was expanded to include the triactinomyxon stage.[4] Similarly, other actinosporeans were folded into the life cycles of various myxosporeans.

Today, the myxozoans, previously thought to be multicellular protozoans, are considered animals by many scientists, though their status has not officially changed. Recent molecular studies suggest they are related to Bilateria or Cnidaria, with Cnidaria being closer morphologically because both groups have extrusive filaments,[5] but with Bilateria being somewhat closer in some genetic studies.[6]

Morphology

Diagram of the structure of a triactionmyxon stage spore of Myxobolus cerebralis

M. cerebralis has many diverse stages ranging from single cells to relatively large spores, not all of which have been studied in detail.

Triactinomyxon stage

The stages that infect fish, called triactinomyxon spores, are made of a single style that is about 150 micrometers (µm) long and three processes or “tails”, each about 200 micrometers long. A sporoplasm packet at the end of the style contains 64 germ cells surrounded by a cellular envelope.[5] There are also three polar capsules, each of which contains a coiled polar filament between 170 and 180 µm long.[2] Polar filaments in both this stage and in the myxospore stage (see picture above) rapidly shoot into the body of the host, creating an opening through which the sporoplasm can enter.

Sporoplasm stage

Upon contact with fish hosts and firing of the polar capsules, the sporoplasm contained within the central style of the triactinomyxon migrates into the epithelium or gut lining. Firstly, this sporoplasm undergoes mitosis to produce more amoeboid cells, which migrate into deeper tissue layers, to reach the cerebral cartilage.[2]

Myxosporean stage

Myxospores, which develop from sporogonic cell stages inside fish hosts, are lenticular. They have a diameter of about 10 micrometers and are made of six cells. Two of these cells form polar capsules, two merge to form a binucleate sporoplasm, and two form protective valves.[5] Myxospores are infective to oligochaetes, and are found among the remains of digested fish cartilage. They are often difficult to distinguish from related species because of morphological similarities across genera. Though M. cerebralis is the only myxosporean ever found in salmonid cartilage, other visually similar species may be present in the skin, nervous system, or muscle.[2]

Life cycle

Life cycle of M. cerebralis

Myxobolus cerebralis has a two-host life cycle involving a salmonid fish and a tubificid oligochaete. So far, the only worm known to be susceptible to M. cerebralis infection is Tubifex tubifex,[2] though what scientists currently call T. tubifex may in fact be more than one species.[3] First, myxospores are ingested by tubificid worms. In the gut lumen of the worm, the spores extrude their polar capsules and attach to the gut epithelium by polar filaments. The shell valves then open along the suture line and the binucleate germ cell penetrates between the intestinal epithelial cells of the worm. This cell multiplies, producing many amoeboid cells by an asexual cell fission process called merogony. As a result of the multiplication process, the intercellular space of the epithelial cells in more than 10 neighbouring worm segments may become infected.[7]

Around 60–90 days postinfection, sexual cell stages of the parasite undergo sporogenesis, and develop into pansporocysts, each of which contains eight triactinomyxon-stage spores. These spores are released from the oligochaete anus into the water.[5] Alternatively, a fish can become infected by eating an infected oligochaete.[5] Infected tubificids can release triactinomyxons for at least a year.[7] The triactinomyxon spores are carried by the water currents, where they can infect a salmonid through the skin. Penetration of the fish by these spores takes only a few seconds. Within five minutes, a sac of germ cells called a sporoplasm has entered the fish epidermis, and within a few hours, the sporoplasm splits into individual cells that will spread through the fish.[5]

Within the fish, both intracellular and extracellular stages reproduce in its cartilage by asexual endogeny, meaning new cells grow from within old cells. The final stage within the fish is the creation of the myxospore, which is formed by sporogony. They are released into the environment when the fish decomposes or is eaten.[5] Some recent research indicates some fish may expel viable myxospores while still alive.[8]

Myxospores are extremely tough: “it was shown that Myxobolus cerebralis spores can tolerate freezing at −20°C for at least 3 months, aging in mud at 13°C for at least 5 months, and passage through the guts of northern pike Esox lucius or mallards Anas platyrhynchos without loss of infectivity” to worms.[9] Triactinomyxons are much shorter-lived, surviving 34 days or less, depending on temperature.[10]

Pathology

Skeletal deformation in a mature brook trout caused by M. cerebralis infection, photo by Dr. Thomas L. Wellborn, Jr.

M. cerebralis infections have been reported from a wide range of salmonid species: eight species of “Atlantic” salmonids, Salmo; four species of “Pacific” salmonids, Oncorhynchus; four species of char, Salvelinus; the grayling, Thymallus thymallus; and the huchen, Hucho hucho.[11] M. cerebralis causes damage to its fish hosts through attachment of triactinomyxon spores and the migrations of various stages through tissues and along nerves, as well as by digesting cartilage.[5] The fish’s tail may darken, but aside from lesions on cartilage, internal organs generally appear healthy.[2] Other symptoms include skeletal deformities and “whirling” behavior (tail-chasing) in young fish, which was thought to have been caused by a loss of equilibrium, but is actually caused by damage to the spinal cord and lower brain stem.[3] Experiments have shown that fish can kill Myxobolus in their skin (possibly using antibodies), but that the fish do not attack the parasites once they have migrated to the central nervous system. This response varies from species to species.[3]

In T. tubifex, the release of triactinomyxon spores from the intestinal wall damages the worm’s mucosa; this may happen thousands of times in a single worm, and is believed to impair nutrient absorption.[5] Also, fish infected with worms have lower body mass and may be discolored.[3] Spores are released from the worm almost exclusively when the temperature is between 10°C and 15°C, so fish in warmer or cooler waters are less likely to be infected, and infection rates vary seasonally.[3]

Susceptibility

Fish size, age, concentration of triactinomyxon spores, and water temperature all affect infection rates in fish, as does the species of the fish in question.[12] The disease has the most impact on fish less than five months old because their skeletons have not ossified. This makes young fish more susceptible to deformities[13] and provides M. cerebralis more cartilage on which to feed.[3] In one study of seven species of many strains, brook trout and rainbow trout (except one strain) were far more heavily affected by M. cerebralis after two hours of exposure than other species were, while bull trout, Chinook salmon, brown trout, and arctic grayling were least severely affected.[12] While brown trout may harbor the parasite, they typically do not show any symptoms, and this species may have been M. cerebralis’ original host.[14] This lack of symptoms in brown trout meant that the parasite was not discovered until after nonnative rainbow trout were introduced in Europe.[3] The susceptibility of various salmonids is listed in Salmonid susceptibility to whirling disease.

Diagnosis

The normally uniform trout cartilage is scarred with lesions in which M. cerebralis spores develop, weakening and deforming the connective tissues.

Moderate or heavy clinical infection of fish with whirling disease can be presumptively diagnosed on the basis of changes in behavior and appearance about 35 to 80 days after initial infection, though “injury or deficiency in dietary tryptophan and ascorbic acid can evoke similar signs”, so conclusive diagnosis may require finding myxospores in the fish’s cartilage.[2] In heavy infections, only examining cartilage microscopically may be needed to find spores.[2] In less severe infections, the most common test involves digestion of the cranial cartilage with the proteases pepsin and trypsin (pepsin-trypsin digest—PTD) before looking for spores. The head and other tissues can be further examined using histopathology to confirm whether the location and morphology of the spores matches what is known for M. cerebralis. Serological identification of spores in tissue sections using an antibody raised against the spores is also possible. Parasite identity can also be confirmed using the polymerase chain reaction to amplify the 415 base pair 18S rRNA gene from M. cerebralis.[15] Fish should be screened at the life stage most susceptible to the parasites. Routine screening using these techniques is carried out in countries where the parasite occurs and in countries such as Australia and Canada that are not known to have the parasite, but where its introduction could threaten local fish.

Impact

M. cerebralis has been reported in Germany (1893), Italy (1954), USSR (1955), including Sakhalin Island (1960), USA (1958), Bulgaria (1960), Yugoslavia (1960), Sweden (1966), South Africa (1966), Scotland (1968), New Zealand (1971), Ecuador (1971), Norway (1971), Colombia (1972), Lebanon (1973), Ireland (1974), Spain (1981) and England (1981)

Although originally a mild pathogen of Salmo trutta in central Europe and other salmonids in northeast Asia, the spread of the rainbow trout (Oncorhynchus mykiss) has greatly increased the impact of this parasite. Having no innate immunity to M. cerebralis, rainbow trout are particularly susceptible, and can release so many spores that even more resistant species in the same area, such as S. trutta, can become overloaded with parasites and incur 80%–90% mortalities. Where M. cerebralis has become well-established, it has caused decline or even elimination of whole cohorts of fish.[16][17]

Impact in Europe

The impact of M. cerebralis in Europe is somewhat lessened because the species is endemic to this region, giving native fish stocks a degree of immunity. Rainbow trout, the most susceptible species to this parasite, are not native to Europe; successfully reproducing feral populations are rare, so few wild rainbow trout are young enough to be susceptible to infection. On the other hand, they are widely reared for restocking sport-fishing waters and for aquaculture, where this parasite has its greatest impact. Hatching and rearing methods designed to prevent infection of rainbow trout fry have proved successful in Europe. These techniques include hatching eggs in spore-free water and rearing fry to the “ossification” stage in tanks or raceways. These methods give particular attention to the quality of water sources to guard against spore introduction during water exchanges.[18] Fry are moved to earthen ponds only when they are considered to be clinically resistant to the parasite, after skeletal ossification occurs.[13]

Impact in New Zealand

M. cerebralis was first found in New Zealand in 1971. The parasite has only been found in rivers in the South Island, away from the most important aquaculture sites. Additionally, salmonid species commercially aquacultured in New Zealand have low susceptibility to whirling disease, and the parasite has also not been shown to affect native salmonids.[19] An important indirect effect of the parasites presence is quarantine restriction placed on exports of salmon products to Australia.

Impact in the United States

M. cerebralis has been reported in nearly two dozen (green) states in the United States, according to the Whirling Disease Initiative

M. cerebralis was first recorded in North America in 1956 in Pennsylvania, having been introduced via infected trout imported from Europe, and has spread steadily south and westwards.[20] Until the 1990s, whirling disease was considered a manageable problem affecting rainbow trout in hatcheries. However, it has recently become established in natural waters of the Rocky Mountain states (Colorado, Wyoming, Utah, Montana, Idaho, New Mexico), where it is causing heavy mortalities in several sportfishing rivers. Some streams in the western United States have lost 90% of their trout.[21] In addition, whirling disease threatens recreational fishing, which is important for the tourism industry, a key component of the economies of some U.S. western states. For example, “the Montana Whirling Disease Task Force estimated trout fishing generated US $300,000,000 in recreational expenditures in Montana alone”.[3] Making matters worse, some of the fishes that M. cerebralis infects (bull trout, cutthroat trout, and steelhead) are already threatened or endangered, and the parasite could worsen their already precarious situations.[3] For reasons that are poorly understood, but probably have to do with environmental conditions, the impact on infected fish has been greatest in Colorado and Montana, and least in California, Michigan, and New York.[22]

Prevention and control

Some biologists have attempted to disarm triactinomyxon spores by making them fire prematurely. In the laboratory, only extreme acidity or basicity, moderate to high concentrations of salts, or electrical current caused premature filament discharge; neurochemicals, cnidarian chemosensitizers, and trout mucus were ineffective,[23] as were anesthetized or dead fish.[24] If spores could be disarmed, they would be unable to infect fish, but it is unclear whether any of the methods that worked in the laboratory could be employed in the wild.

Some strains of fish are more resistant than others, even within species;[12] using resistant strains may help reduce the incidence and severity of whirling disease in aquaculture. There is also some circumstantial evidence that fish populations can develop resistance to the disease over time.[25] Additionally, aquaculturists may avoid M. cerebralis infections by not using earthen ponds for raising young fish; this keeps them away from possibly infected tubificids and makes it easier to eliminate spores and oligochaetes through filtration, chlorination, and ultraviolet bombardment.[2] To minimise tubificid populations, techniques include periodic disinfection of the hatchery or aquaculture ponds, and the rearing of small trout indoors in pathogen-free water. Smooth-faced concrete or plastic-lined raceways that are kept clean and free of contaminated water keep aquaculture facilities free of the disease.[2]

Lastly, some drugs, such as furazolidone, furoxone, benomyl, fumagillin, proguanil and clamoxyquine, have been shown to impede spore development, which reduces infection rates.[2] For example, one study showed that feeding fumagillin to O. mykiss reduced the number of infected fish from between 73% and 100% to between 10% and 20%.[9] Unfortunately, this treatment is considered unsuitable for wild trout populations,[7] and no drug treatment has ever been shown to be effective in the studies required for United States Food and Drug Administration approval.[3]

Recreational and sports fishers can help to prevent the spread of the parasite in a number of ways. Cleaning fishing equipment between fishing trips and never transporting fish from one body of water to another should protect against cross-contamination of waterways. Spores are particularly persistent in felt-soled wading shoes, which should be treated with 10% chlorine bleach and water for at least 15 minutes and then rinsed thoroughly. Fish bones or entrails should never be disposed of in any body of water, since spores from the carcass will be released into the waterway. Salmon and trout should not be used as bait.

See also

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Greetings all Constitution loving Americans, my name is Wolf.
Watch this 3-minute video by news media KBZK which can also be seen at KBZK.com or by following this link ( http://www.kbzk.com/news/citizens-thr… /?fb_comment_id=fbc_698858186832403_9371

­8674_699210960130459#!prettyPhoto/0/ ) in the description. I wanted to effect a citizen’s arrest justice of the peace Rick West, which is governed by MCA 46-6-502. I contacted the US Marshalls office, the Montana Attorney General’s office twice, spoke with Marty Lambert who is the Gallatin County District Attorney and the FBI. In a meeting with the FBI on Friday January 31 at 10 AM , I explained my knowledge of the citizen’s arrest laws and that I had already contacted the US Marshall to be available to take Rick West into custody for violation of U.S. Code Title 18 Chapter 13 Section 241. I am linking the code here for you so you can read it yourself. You need to know this code as it is your only protection. ( http://uscode.house.gov/view.xhtml?re… ) My phone and email records will are proof of my extensive contact with government agencies to insure that I followed all the laws of a citizen’s arrest. You may detain a person after a citizen’s arrest but you must turn them over immediately to the nearest law agent. As this crime was a federal crime, I had contacted the US Marshall to be there to take custody. This was all explained to the FBI and the Sheriff before his interview with that media.
Yet how does Sheriff Gootkin describe us, just listen in the interview, now
The Sheriff says we threatened and talked of kidnapping, which caused Rick West’s family to live in fear. How disgusting of a lie is that? He is charged with keeping the peace not instilling fear. However, he tries to instill fear in the People by making unfounded accusations. Now we all know that if I had threated to kidnap a judge to the FBI, I would be in jail.
Then he compares concerned Americans who only want the law to be equally applied, to people who write bad check and of all things WHIRLING DISEASE, which is a parasitic, crippling disease. I have included a link so you can read about it as well. ( http://en.wikipedia.org/wiki/Myxobolu… ). Yes folks, people who want to hold our law agencies, courts, and elected offices to the same standards the People are held to are referred to as a disease. You decide folks, is this the kind of law agent you want in your country? Comment on the KBZK web page and if you outraged enough write the Gallatin County Commissioners and demand a Citizens Tribunal according to MCA 2-2-144 (5). ( http://www.gallatin.mt.gov/Public_Doc… )
Follow us at http://mtpatriot.net/

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